Over-expression of miR-34c leads to early-life visceral fat

Welcome to Huang Lab - Publications, Huang Lab

Summary diagram for proposed effects of AMPK, SIRT1, PPARγ, and

Over-expression of miR-34c leads to early-life visceral fat accumulation and insulin resistance

Frontiers Characteristic MicroRNAs Linked to Dysregulated Metabolic Pathways in Qatari Adult Subjects With Obesity and Metabolic Syndrome

Adipocyte precursor cells from first degree relatives of type 2 diabetic patients feature changes in hsa‐mir‐23a‐5p, ‐193a‐5p, and ‐193b‐5p and insulin‐like growth factor 2 expression - Mirra - 2021 - The FASEB

Brian Deng's Instagram, Twitter & Facebook on IDCrawl

Overexpression of miR-34a-resistant SIRT1 or MDM4 does not inhibit

MicroRNAs: a crossroad that connects obesity to immunity and aging, Immunity & Ageing

Effect of diet and genotype on the miRNome of mice with altered lipoprotein metabolism: iScience

PDF) MiRNA-34c Regulates Bovine Sertoli Cell Proliferation, Gene

Catch-up growth in juvenile rats, fat expansion, and dysregulation

Baseline characteristics and ultrasonographic findings of

A mouse model for a partially inactive obesity-associated human

CACUL1 reciprocally regulates SIRT1 and LSD1 to repress PPARγ and